We consider four mechanisms by which apparent discontinuities in the distribution of antisociality could arise: (1) executive genes or hormonal systems, (2) multiplicative interactions of predisposing factors, (3) environmental tracking into a limited number of social roles, and (4) cross-generational gene-environment interactions. A more explicit consideration of complex self-organizing dynamic systems may help us understand the maintenance of antisocial subpopulations.
|Original language||English (US)|
|Number of pages||2|
|Journal||Behavioral and Brain Sciences|
|Publication status||Published - 1995|
ASJC Scopus subject areas
- Ecology, Evolution, Behavior and Systematics
- Neuropsychology and Physiological Psychology